Brandon J. Biesiadecki

Brandon J. Biesiadecki

Department of Physiology and Cell Biology, The Ohio State University

Affiliated withThe Ohio State University

Research Area

Biography

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JoVE Journal Publications

ArticleTotal : 1
Year
A Simple and Effective Method to Consistently Isolate Mouse Cardiomyocytes
Publication title

Cited by 1

2022

Other Publications

Article
Year
Increased phosphorylation of tropomyosin, troponin I, and myosin light chain-2 after stretch in rabbit ventricular myocardium under physiological conditions.

Journal of molecular and cellular cardiology| PubMed ID: 20298699

2010
2011
2012
2012
2012
2012
2012
2012
Effects of increased systolic Ca(2+) and β-adrenergic stimulation on Ca(2+) transient decline in NOS1 knockout cardiac myocytes.

Nitric oxide : biology and chemistry / official journal of the Nitric Oxide Society| PubMed ID: 22960389

2012
Tropomyosin Ser-283 pseudo-phosphorylation slows myofibril relaxation.

Archives of biochemistry and biophysics| PubMed ID: 23232082

2013
2014
2014
2014
2014
2014
2014
2015
2015
Molecular Mapping of Sinoatrial Node HCN Channel Expression in the Human Heart.

Circulation. Arrhythmia and electrophysiology| PubMed ID: 26304511

2015
2015
Should we treat heart failure with phosphatase inhibitors? Better to start at the end.

Journal of molecular and cellular cardiology| PubMed ID: 26497613

2015
2016
Myofilament modulation of contraction.

Archives of biochemistry and biophysics| PubMed ID: 27156968

2016
2016
2016
2016
2016
2014
Functional communication between PKC-targeted cardiac troponin I phosphorylation sites.

Archives of biochemistry and biophysics| PubMed ID: 28587770

2017
2017
2018
2019
Troponin I modulation of cardiac performance: Plasticity in the survival switch.

Archives of biochemistry and biophysics| PubMed ID: 30684464

2019
Current and Future Directions of Myofilament Regulation.

Archives of biochemistry and biophysics| PubMed ID: 31051122

2019
2020
2020
mutations in the tropomyosin binding region of TNT1 disrupt its role in contractile inhibition and stimulate cardiac dysfunction.

Proceedings of the National Academy of Sciences of the United States of America| PubMed ID: 32690703

2020
2020
2022