Kelly A. Avery-Kiejda Medical Genetics Hunter Medical Research Institute Biography Publications Institution JoVE Articles Kelly A. Avery-Kiejda has not added a biography. If you are Kelly A. Avery-Kiejda and would like to personalize this page please email our Author Liaison for assistance. Publications The Intron 3 16 bp Duplication Polymorphism of P53 (rs17878362) is Not Associated with Increased Risk of Developing Triple-negative Breast Cancer Breast Cancer Research and Treatment. Nov, 2018 | Pubmed ID: 30430302 A Novel Polymorphic Repeat in the Upstream Regulatory Region of the Estrogen-induced Gene EIG121 is Not Associated with the Risk of Developing Breast or Endometrial Cancer BMC Research Notes. Month, 2016 | Pubmed ID: 27230222 A Polymorphic Repeat in the IGF1 Promoter Influences the Risk of Endometrial Cancer Endocrine Connections. May, 2016 | Pubmed ID: 27090263 The Presence of the Intron 3 16 Bp Duplication Polymorphism of P53 (rs17878362) in Breast Cancer is Associated with a Low Δ40p53:p53 Ratio and Better Outcome Carcinogenesis. Jan, 2016 | Pubmed ID: 26586794 MiRNAs and Other Epigenetic Changes As Biomarkers in Triple Negative Breast Cancer International Journal of Molecular Sciences. Month, 2015 | Pubmed ID: 26633365 Novel Genes Associated with Lymph Node Metastasis in Triple Negative Breast Cancer Scientific Reports. Month, 2015 | Pubmed ID: 26537449 Proteotranscriptomic Profiling of 231-BR Breast Cancer Cells: Identification of Potential Biomarkers and Therapeutic Targets for Brain Metastasis Molecular & Cellular Proteomics : MCP. Sep, 2015 | Pubmed ID: 26041846 Methylome Sequencing in Triple-negative Breast Cancer Reveals Distinct Methylation Clusters with Prognostic Value Nature Communications. Month, 2015 | Pubmed ID: 25641231 The Expression of Dicer and Drosha in Matched Normal Tissues, Tumours and Lymph Node Metastases in Triple Negative Breast Cancer BMC Cancer. Month, 2014 | Pubmed ID: 24725360 Decreased Expression of Key Tumour Suppressor MicroRNAs is Associated with Lymph Node Metastases in Triple Negative Breast Cancer BMC Cancer. Month, 2014 | Pubmed ID: 24479446 The Relative MRNA Expression of P53 Isoforms in Breast Cancer is Associated with Clinical Features and Outcome Carcinogenesis. Mar, 2014 | Pubmed ID: 24336193 Low Prevalence of Germline PALB2 Mutations in Australian Triple-negative Breast Cancer International Journal of Cancer. Jan, 2014 | Pubmed ID: 23824750 STaRRRT: a Table of Short Tandem Repeats in Regulatory Regions of the Human Genome BMC Genomics. Month, 2013 | Pubmed ID: 24228761 Regulators of Global Genome Repair Do Not Respond to DNA Damaging Therapy but Correlate with Survival in Melanoma PloS One. Month, 2013 | Pubmed ID: 23940574 P53 in Human Melanoma Fails to Regulate Target Genes Associated with Apoptosis and the Cell Cycle and May Contribute to Proliferation BMC Cancer. Month, 2011 | Pubmed ID: 21615965 BRIP1, PALB2, and RAD51C Mutation Analysis Reveals Their Relative Importance As Genetic Susceptibility Factors for Breast Cancer Breast Cancer Research and Treatment. Jun, 2011 | Pubmed ID: 21409391 Nucleotide Excision Repair Gene Expression After Cisplatin Treatment in Melanoma Cancer Research. Oct, 2010 | Pubmed ID: 20807809 Glucose-regulated Protein 78 Antagonizes Cisplatin and Adriamycin in Human Melanoma Cells Carcinogenesis. Feb, 2009 | Pubmed ID: 18842681 Up-regulation of Mcl-1 is Critical for Survival of Human Melanoma Cells Upon Endoplasmic Reticulum Stress Cancer Research. Aug, 2008 | Pubmed ID: 18701495 Small Molecular Weight Variants of P53 Are Expressed in Human Melanoma Cells and Are Induced by the DNA-damaging Agent Cisplatin Clinical Cancer Research : an Official Journal of the American Association for Cancer Research. Mar, 2008 | Pubmed ID: 18310316 Activation of Jun N-terminal Kinase is a Mediator of Vincristine-induced Apoptosis of Melanoma Cells Anti-cancer Drugs. Feb, 2008 | Pubmed ID: 18176116 Een eenvoudige migratie/invasie Workflow met behulp van een geautomatiseerde Live-cel Imager Xiajie Zhang1,2, Brianna C. Morten1,2, Rodney J. Scott1,2,3, Kelly A. Avery-Kiejda1,2 1Medical Genetics, Hunter Medical Research Institute, 2Priority Research Centre for Cancer Research, Innovation and Translation, School of Biomedical Sciences and Pharmacy, Faculty of Health and Medicine, University of Newcastle, 3Pathology North, John Hunter Hospital JoVE 59042 Cancer Research Vergelijking van de drie verschillende methoden voor het bepalen van de cel proliferatie in borstkanker cellijnen Brianna C. Morten1,2, Rodney J. Scott1,2,3, Kelly A. Avery-Kiejda1,2 1Medical Genetics, Hunter Medical Research Institute, 2Priority Research Centre for Cancer, School of Biomedical Sciences and Pharmacy, Faculty of Health and Medicine, University of Newcastle, 3Pathology North, John Hunter Hospital Biology
Een eenvoudige migratie/invasie Workflow met behulp van een geautomatiseerde Live-cel Imager Xiajie Zhang1,2, Brianna C. Morten1,2, Rodney J. Scott1,2,3, Kelly A. Avery-Kiejda1,2 1Medical Genetics, Hunter Medical Research Institute, 2Priority Research Centre for Cancer Research, Innovation and Translation, School of Biomedical Sciences and Pharmacy, Faculty of Health and Medicine, University of Newcastle, 3Pathology North, John Hunter Hospital JoVE 59042 Cancer Research
Vergelijking van de drie verschillende methoden voor het bepalen van de cel proliferatie in borstkanker cellijnen Brianna C. Morten1,2, Rodney J. Scott1,2,3, Kelly A. Avery-Kiejda1,2 1Medical Genetics, Hunter Medical Research Institute, 2Priority Research Centre for Cancer, School of Biomedical Sciences and Pharmacy, Faculty of Health and Medicine, University of Newcastle, 3Pathology North, John Hunter Hospital Biology