Charles Krieger Department of Biomedical Physiology and Kinesiology Simon Fraser University Biography Publications Institution JoVE Articles Charles Krieger has not added a biography. If you are Charles Krieger and would like to personalize this page please email our Author Liaison for assistance. Publications Submyeloablative Conditioning with Busulfan Permits Bone Marrow-derived Cell Accumulation in a Murine Model of Alzheimer's Disease Neuroscience Letters. Jan, 2015 | Pubmed ID: 25582787 Phospho-regulated Drosophila Adducin is a Determinant of Synaptic Plasticity in a Complex with Dlg and PIP2 at the Larval Neuromuscular Junction Biology Open. 2014 | Pubmed ID: 25416060 Motoneuron Firing in Amyotrophic Lateral Sclerosis (ALS) Frontiers in Human Neuroscience. 2014 | Pubmed ID: 25294995 Activity-dependent Secretion of Progranulin from Synapses Journal of Cell Science. Dec, 2013 | Pubmed ID: 24046442 Ethical Considerations in the Management of Amyotrophic Lateral Sclerosis Progress in Neurobiology. Nov, 2013 | Pubmed ID: 23735671 Myelosuppressive Conditioning Using Busulfan Enables Bone Marrow Cell Accumulation in the Spinal Cord of a Mouse Model of Amyotrophic Lateral Sclerosis PloS One. 2013 | Pubmed ID: 23593276 The Neuroinflammatory Response in ALS: The Roles of Microglia and T Cells Neurology Research International. 2012 | Pubmed ID: 22666587 Reduced Protein O-glycosylation in the Nervous System of the Mutant SOD1 Transgenic Mouse Model of Amyotrophic Lateral Sclerosis Neuroscience Letters. May, 2012 | Pubmed ID: 22521585 Clinical and Pathological Features of Amyotrophic Lateral Sclerosis Caused by Mutation in the C9ORF72 Gene on Chromosome 9p Acta Neuropathologica. Mar, 2012 | Pubmed ID: 22228244 单核细胞浸润触发 EAE 级数,但无助到驻地小胶质细胞池。 Nature Neuroscience. Sep, 2011 | Pubmed ID: 21804537 果蝇内收蛋白调节 Dlg 磷酸化和 Dlg 的靶向的突触和上皮膜。 Developmental Biology. Sep, 2011 | Pubmed ID: 21791202 A Second ALS Patient Having an L67P Mutation in Exon 3 of the Cu/Zn Superoxide Dismutase Gene Amyotrophic Lateral Sclerosis : Official Publication of the World Federation of Neurology Research Group on Motor Neuron Diseases. Nov, 2011 | Pubmed ID: 21692663 Fus Gene Mutations in Familial and Sporadic Amyotrophic Lateral Sclerosis Muscle & Nerve. Aug, 2010 | Pubmed ID: 20544928 Mislocalization of TDP-43 in the G93A Mutant SOD1 Transgenic Mouse Model of ALS Neuroscience Letters. Jul, 2009 | Pubmed ID: 19379791 骨髓细胞在中枢神经系统的小鼠模型的肌萎缩侧索硬化与血管相关联,并表示 CX (3) CR1。 Glia. Oct, 2009 | Pubmed ID: 19243075 Pilot Study of Granulocyte Colony Stimulating Factor (G-CSF)-mobilized Peripheral Blood Stem Cells in Amyotrophic Lateral Sclerosis (ALS) Muscle & Nerve. May, 2008 | Pubmed ID: 18335482 Neuromuscular Dysfunction in the Mutant Superoxide Dismutase Mouse Model of Amyotrophic Lateral Sclerosis Amyotrophic Lateral Sclerosis : Official Publication of the World Federation of Neurology Research Group on Motor Neuron Diseases. 2008 | Pubmed ID: 18273716 本地自我更新可以维持中枢神经系统小胶质细胞维护和整个成年生活的功能。 Nature Neuroscience. Dec, 2007 | Pubmed ID: 18026097 起源和骨髓细胞在肌萎缩侧索硬化的模型小鼠中枢神经系统的分布。 Glia. May, 2006 | Pubmed ID: 16518833 Overexpressed Mutant G93A Superoxide Dismutase Protects Calcineurin from Inactivation Brain Research. Molecular Brain Research. Jun, 2004 | Pubmed ID: 15193434 Chronically Implanted Epineural Electrodes for Repeated Assessment of Nerve Conduction Velocity and Compound Action Potential Amplitude in Rodents Journal of Neuroscience Methods. Jan, 2004 | Pubmed ID: 14687672 Aberrant Protein Kinases and Phosphoproteins in Amyotrophic Lateral Sclerosis Trends in Pharmacological Sciences. Oct, 2003 | Pubmed ID: 14559406 Protein Kinase and Protein Phosphatase Expression in the Central Nervous System of G93A MSOD Over-expressing Mice Journal of Neurochemistry. Apr, 2003 | Pubmed ID: 12675918 Lumbar Motoneuron Fate in a Mouse Model of Amyotrophic Lateral Sclerosis Neuroreport. Dec, 2002 | Pubmed ID: 12488813 Protein Phosphorylation Networks in Motor Neuron Death Progress in Drug Research. Fortschritte Der Arzneimittelforschung. Progrès Des Recherches Pharmaceutiques. 2002 | Pubmed ID: 12458964 Mitochondria, Ca2+ and Neurodegenerative Disease European Journal of Pharmacology. Jul, 2002 | Pubmed ID: 12151010 白消安作为骨髓抑制剂在小鼠生成稳定的高水平骨髓嵌合体 Kyle Peake1, John Manning1, Coral-Ann Lewis1,2, Christine Barr1, Fabio Rossi2, Charles Krieger1,3 1Department of Biomedical Physiology and Kinesiology, Simon Fraser University, 2The Biomedical Research Centre, University of British Columbia, 3Division of Neurology, Department of Medicine, Neuromuscular Disease Unit, VHHSC Medicina 检测 Simon Wang1, SooHyun Yoo1, Hae-yoon Kim1, Mannan Wang2, Clare Zheng2, Wade Parkhouse2, Charles Krieger2, Nicholas Harden1 1Department of Molecular Biology and Biochemistry, Simon Fraser University, 2Department of Biomedical Physiology and Kinesiology, Simon Fraser University JoVE 52139 Neuroscienze
白消安作为骨髓抑制剂在小鼠生成稳定的高水平骨髓嵌合体 Kyle Peake1, John Manning1, Coral-Ann Lewis1,2, Christine Barr1, Fabio Rossi2, Charles Krieger1,3 1Department of Biomedical Physiology and Kinesiology, Simon Fraser University, 2The Biomedical Research Centre, University of British Columbia, 3Division of Neurology, Department of Medicine, Neuromuscular Disease Unit, VHHSC Medicina
检测 Simon Wang1, SooHyun Yoo1, Hae-yoon Kim1, Mannan Wang2, Clare Zheng2, Wade Parkhouse2, Charles Krieger2, Nicholas Harden1 1Department of Molecular Biology and Biochemistry, Simon Fraser University, 2Department of Biomedical Physiology and Kinesiology, Simon Fraser University JoVE 52139 Neuroscienze