Syam Nair Centre of Perinatal Medicine and Health, Institute of Clinical Sciences, Department of Obstetrics and Gynecology, Sahlgrenska Academy University of Gothenburg Biography Publications Institution JoVE Articles Syam Nair has not added a biography. If you are Syam Nair and would like to personalize this page please email our Author Liaison for assistance. Publications Silencing of STE20-type Kinase TAOK1 Confers Protection Against Hepatocellular Lipotoxicity Through Metabolic Rewiring Hepatology Communications. Apr, 2023 | Pubmed ID: 36930872 Inhibition of MAP4K4 Signaling Initiates Metabolic Reprogramming to Protect Hepatocytes from Lipotoxic Damage Journal of Lipid Research. Jul, 2022 | Pubmed ID: 35679904 Induction of Mitochondrial Fragmentation and Mitophagy After Neonatal Hypoxia-Ischemia Cells. Apr, 2022 | Pubmed ID: 35406757 Single-cell Atlas Reveals Meningeal Leukocyte Heterogeneity in the Developing Mouse Brain Genes & Development. Aug, 2021 | Pubmed ID: 34301765 STE20-Type Protein Kinase MST4 Controls NAFLD Progression by Regulating Lipid Droplet Dynamics and Metabolic Stress in Hepatocytes Hepatology Communications. Jul, 2021 | Pubmed ID: 34278168 Galectin-3 Modulates Microglia Inflammation in Vitro but Not Neonatal Brain Injury in Vivo Under Inflammatory Conditions Developmental Neuroscience. 2021 | Pubmed ID: 34130282 Neuroprotection Offered by Mesenchymal Stem Cells in Perinatal Brain Injury: Role of Mitochondria, Inflammation, and Reactive Oxygen Species Journal of Neurochemistry. 07, 2021 | Pubmed ID: 33314066 Plin2-deficiency Reduces Lipophagy and Results in Increased Lipid Accumulation in the Heart Scientific Reports. May, 2019 | Pubmed ID: 31061399 The Role of Mitochondrial and Endoplasmic Reticulum Reactive Oxygen Species Production in Models of Perinatal Brain Injury Antioxidants & Redox Signaling. Sep, 2019 | Pubmed ID: 30957515 Lipopolysaccharide-induced Alteration of Mitochondrial Morphology Induces a Metabolic Shift in Microglia Modulating the Inflammatory Response in Vitro and in Vivo Glia. Jun, 2019 | Pubmed ID: 30637805 Neuroprotective Exendin-4 Enhances Hypothermia Therapy in a Model of Hypoxic-ischaemic Encephalopathy Brain : a Journal of Neurology. Oct, 2018 | Pubmed ID: 30165597 Mitochondrial Dynamics, Mitophagy and Biogenesis in Neonatal Hypoxic-ischaemic Brain Injury FEBS Letters. Mar, 2018 | Pubmed ID: 29265370 Cell Death in the Developing Brain After Hypoxia-Ischemia Frontiers in Cellular Neuroscience. 2017 | Pubmed ID: 28878624 Mitochondria, Bioenergetics and Excitotoxicity: New Therapeutic Targets in Perinatal Brain Injury Frontiers in Cellular Neuroscience. 2017 | Pubmed ID: 28747873 Systemic Activation of Toll-like Receptor 2 Suppresses Mitochondrial Respiration and Exacerbates Hypoxic-ischemic Injury in the Developing Brain Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism. Apr, 2017 | Pubmed ID: 28139935 Temporal Characterization of Microglia/Macrophage Phenotypes in a Mouse Model of Neonatal Hypoxic-Ischemic Brain Injury Frontiers in Cellular Neuroscience. 2016 | Pubmed ID: 28018179 The Effect of Osteopontin and Osteopontin-derived Peptides on Preterm Brain Injury Journal of Neuroinflammation. Dec, 2014 | Pubmed ID: 25465048 후안와 정맥 주입의 신생아 설치류 모델 Eridan Rocha-Ferreira1, Syam Nair1, Owen Herrock1, Erik Axel Andersson2, Carl Joakim Ek2, Carina Mallard2, Henrik Hagberg1 1Centre of Perinatal Medicine and Health, Institute of Clinical Sciences, Department of Obstetrics and Gynecology, Sahlgrenska Academy, University of Gothenburg, 2Centre of Perinatal Medicine and Health, Institute of Neuroscience and Physiology, Sahlgrenska Academy, University of Gothenburg JoVE 65386 Neuroscienze
후안와 정맥 주입의 신생아 설치류 모델 Eridan Rocha-Ferreira1, Syam Nair1, Owen Herrock1, Erik Axel Andersson2, Carl Joakim Ek2, Carina Mallard2, Henrik Hagberg1 1Centre of Perinatal Medicine and Health, Institute of Clinical Sciences, Department of Obstetrics and Gynecology, Sahlgrenska Academy, University of Gothenburg, 2Centre of Perinatal Medicine and Health, Institute of Neuroscience and Physiology, Sahlgrenska Academy, University of Gothenburg JoVE 65386 Neuroscienze