Elsa N. Bou Ghanem Department of Microbiology and Immunology University at Buffalo School of Medicine Biography Publications Institution JoVE Articles Elsa N. Bou Ghanem has not added a biography. If you are Elsa N. Bou Ghanem and would like to personalize this page please email our Author Liaison for assistance. Publications The Age-Driven Decline in Neutrophil Function Contributes to the Reduced Efficacy of the Pneumococcal Conjugate Vaccine in Old Hosts Frontiers in Cellular and Infection Microbiology. 2022 | Pubmed ID: 35402289 A Murine Model for Enhancement of Streptococcus Pneumoniae Pathogenicity Upon Viral Infection and Advanced Age Infection and Immunity. 07, 2021 | Pubmed ID: 34031128 Older but Not Wiser: the Age-Driven Changes in Neutrophil Responses During Pulmonary Infections Infection and Immunity. 03, 2021 | Pubmed ID: 33495271 Testing Anti-Pneumococcal Antibody Function Using Bacteria and Primary Neutrophils Methods in Molecular Biology (Clifton, N.J.). 2021 | Pubmed ID: 32959268 Extracellular Adenosine Signaling Reverses the Age-driven Decline in the Ability of Neutrophils to Kill Streptococcus Pneumoniae Aging Cell. 10, 2020 | Pubmed ID: 32790148 Neutrophils Are Required During Immunization With the Pneumococcal Conjugate Vaccine for Protective Antibody Responses and Host Defense Against Infection The Journal of Infectious Diseases. 09, 2020 | Pubmed ID: 32391562 Extracellular Adenosine Enhances the Ability of PMNs to Kill Streptococcus Pneumoniae by Inhibiting IL-10 Production Journal of Leukocyte Biology. 09, 2020 | Pubmed ID: 32017200 Pneumolysin Induces 12-Lipoxygenase-Dependent Neutrophil Migration During Infection Journal of Immunology (Baltimore, Md. : 1950). 01, 2020 | Pubmed ID: 31776202 A1 Adenosine Receptor Signaling Reduces Streptococcus Pneumoniae Adherence to Pulmonary Epithelial Cells by Targeting Expression of Platelet-activating Factor Receptor Cellular Microbiology. 02, 2020 | Pubmed ID: 31709673 MSphere of Influence: Adenosine in Host Defense Against Bacterial Pneumonia-Friend or Foe? MSphere. 07, 2019 | Pubmed ID: 31292232 PepN is a Non-essential, Cell Wall-localized Protein That Contributes to Neutrophil Elastase-mediated Killing of Streptococcus Pneumoniae PloS One. 2019 | Pubmed ID: 30707714 Nasopharyngeal Exposure to Induces Extended Age-Dependent Protection Against Pulmonary Infection Mediated by Antibodies and CD138 Cells Journal of Immunology (Baltimore, Md. : 1950). 06, 2018 | Pubmed ID: 29661828 InlA Favorise La Diffusion De Listeria Monocytogenes Dans Les Ganglions Lymphatiques Mésentériques Au Cours De L'infection Des Souris D'origine Alimentaire PLoS Pathogens. 2012 | Pubmed ID: 23166492 Des Cellules T CD8 + Affichent Une Aptitude Différentielle à Activation Spectateur Axée Sur Les Cytokines Cellular Immunology. 2011 | Pubmed ID: 21978649 Facteurs Intrinsèques Des Lymphocytes T Contribuent à La Capacité Différentielle Des Cellules T CD8 + Rapidement Sécrètent IFN-γ En L'absence D'antigène Journal of Immunology (Baltimore, Md. : 1950). Feb, 2011 | Pubmed ID: 21191063 Plusieurs Mécanismes Contribuent à La Réponse De L'interféron Gamma Rapide Robuste Par Les Cellules T CD8 + Au Cours De L'infection à Listeria Monocytogenes Infection and Immunity. Apr, 2009 | Pubmed ID: 19179413 Un modèle murin pour la transition de Streptococcus pneumoniae de colonisateur à pathogène lors d’une co-infection virale récapitule la maladie exacerbée par l’âge Alexsandra Lenhard*1, Basma H. Joma*2,3, Nalat Siwapornchai2, Anders P. Hakansson4, John M. Leong2,5, Elsa N. Bou Ghanem1 1Department of Microbiology and Immunology, University at Buffalo School of Medicine, 2Department of Molecular Biology and Microbiology, Tufts University School of Medicine, 3Graduate Program in Immunology, Tufts Graduate School of Biomedical Sciences, 4Department of Translational Medicine, Lund University, 5Stuart B. Levy Center for the Integrated Management of Antimicrobial Resistance, Tufts University JoVE 64419 면역학 및 감염병학 La transmission orale de Elsa N. Bou Ghanem1, Tanya Myers-Morales1, Grant S. Jones1, Sarah E.F. D'Orazio1 1Department of Microbiology, Immunology, and Molecular Genetics, University of Kentucky JoVE 50381 면역학 및 감염병학
Un modèle murin pour la transition de Streptococcus pneumoniae de colonisateur à pathogène lors d’une co-infection virale récapitule la maladie exacerbée par l’âge Alexsandra Lenhard*1, Basma H. Joma*2,3, Nalat Siwapornchai2, Anders P. Hakansson4, John M. Leong2,5, Elsa N. Bou Ghanem1 1Department of Microbiology and Immunology, University at Buffalo School of Medicine, 2Department of Molecular Biology and Microbiology, Tufts University School of Medicine, 3Graduate Program in Immunology, Tufts Graduate School of Biomedical Sciences, 4Department of Translational Medicine, Lund University, 5Stuart B. Levy Center for the Integrated Management of Antimicrobial Resistance, Tufts University JoVE 64419 면역학 및 감염병학
La transmission orale de Elsa N. Bou Ghanem1, Tanya Myers-Morales1, Grant S. Jones1, Sarah E.F. D'Orazio1 1Department of Microbiology, Immunology, and Molecular Genetics, University of Kentucky JoVE 50381 면역학 및 감염병학