Marc Freichel Pharmakologisches Institut Ruprecht-Karls-Universität Heidelberg Biography Publications Institution JoVE Articles Marc Freichel has not added a biography. If you are Marc Freichel and would like to personalize this page please email our Author Liaison for assistance. Publications Development of an AAV9-RNAi-mediated Silencing Strategy to Abrogate TRPM4 Expression in the Adult Heart Pflugers Archiv : European Journal of Physiology. Mar, 2021 | Pubmed ID: 33580817 Transcriptional Signatures Regulated by TRPC1/C4-mediated Background Ca Entry After Pressure-overload Induced Cardiac Remodelling Progress in Biophysics and Molecular Biology. 01, 2021 | Pubmed ID: 32738354 Genetic Background Influences Expression and Function of the Cation Channel TRPM4 in the Mouse Heart Basic Research in Cardiology. 11, 2020 | Pubmed ID: 33205255 Control of Insulin Release by Transient Receptor Potential Melastatin 3 (TRPM3) Ion Channels Cellular Physiology and Biochemistry : International Journal of Experimental Cellular Physiology, Biochemistry, and Pharmacology. Nov, 2020 | Pubmed ID: 33166100 L-type Blocker STIMulate Ca Entry in Synthetic VSMCs Cell Calcium. 11, 2020 | Pubmed ID: 32892148 Cardiomyocyte-Specific Deletion of Orai1 Reveals Its Protective Role in Angiotensin-II-Induced Pathological Cardiac Remodeling Cells. 04, 2020 | Pubmed ID: 32354146 Analysis of Mrgprb2 Receptor-Evoked Ca Signaling in Bone Marrow Derived (BMMC) and Peritoneal (PMC) Mast Cells of TRPC-Deficient Mice Frontiers in Immunology. 2020 | Pubmed ID: 32322252 Angiotensin-II-Evoked Ca Entry in Murine Cardiac Fibroblasts Does Not Depend on TRPC Channels Cells. 01, 2020 | Pubmed ID: 32013125 Assessment of PEEP-Ventilation and the Time Point of Parallel-Conductance Determination for Pressure-Volume Analysis Under β-Adrenergic Stimulation in Mice Frontiers in Cardiovascular Medicine. 2019 | Pubmed ID: 31111037 Methylglyoxal Evokes Acute Ca Transients in Distinct Cell Types and Increases Agonist-evoked Ca Entry in Endothelial Cells Via CRAC Channels Cell Calcium. 03, 2019 | Pubmed ID: 30658323 Compensatory Mechanisms for Methylglyoxal Detoxification in Experimental & Clinical Diabetes Molecular Metabolism. 12, 2018 | Pubmed ID: 30287091 Deletion of Orai2 Augments Endogenous CRAC Currents and Degranulation in Mast Cells Leading to Enhanced Anaphylaxis Cell Calcium. May, 2018 | Pubmed ID: 29604961 TRPC Proteins Contribute to Development of Diabetic Retinopathy and Regulate Glyoxalase 1 Activity and Methylglyoxal Accumulation Molecular Metabolism. Mar, 2018 | Pubmed ID: 29373286 Heteromeric Channels Formed by TRPC1, TRPC4 and TRPC5 Define Hippocampal Synaptic Transmission and Working Memory The EMBO Journal. 09, 2017 | Pubmed ID: 28790178 Dicarbonyls and Advanced Glycation End-Products in the Development of Diabetic Complications and Targets for Intervention International Journal of Molecular Sciences. May, 2017 | Pubmed ID: 28475116 TRPM4-mediated Control of FcεRI-evoked Ca(2+) Elevation Comprises Enhanced Plasmalemmal Trafficking of TRPM4 Channels in Connective Tissue Type Mast Cells Scientific Reports. 09, 2016 | Pubmed ID: 27624684 A Background Ca2+ Entry Pathway Mediated by TRPC1/TRPC4 is Critical for Development of Pathological Cardiac Remodelling European Heart Journal. Sep, 2015 | Pubmed ID: 26069213 Adenylyl Cyclase-mediated Effects Contribute to Increased Isoprenaline-induced Cardiac Contractility in TRPM4-deficient Mice Journal of Molecular and Cellular Cardiology. Sep, 2014 | Pubmed ID: 24972051 TRPC4- and TRPC4-containing Channels Handbook of Experimental Pharmacology. 2014 | Pubmed ID: 24756704 The Role of CGMP/cGKI Signalling and Trpc Channels in Regulation of Vascular Tone Cardiovascular Research. Nov, 2013 | Pubmed ID: 23832809 Canonical Transient Receptor Channel 5 (TRPC5) and TRPC1/4 Contribute to Seizure and Excitotoxicity by Distinct Cellular Mechanisms Molecular Pharmacology. Feb, 2013 | Pubmed ID: 23188715 通过压力-体积环路分析确定心脏对β肾上腺素能刺激的反应 Rebekka Medert*1,2, Lucas Bacmeister*1,2,3, Sebastian Segin*1,2,4, Marc Freichel1,2, Juan E. Camacho Londoño1,2 1Pharmakologisches Institut, Ruprecht-Karls-Universität Heidelberg, 2DZHK (German Centre for Cardiovascular Research), partner site Heidelberg/Mannheim, 3Department of Internal Medicine III, University of Heidelberg, 4Department of Anesthesiology, University Hospital RWTH Aachen JoVE 62057 의학 腹膜源性肥大细胞的分离及其功能特征的 Ca2 +成像和脱粒测定 Volodymyr Tsvilovskyy1, Alejandra Solis-Lopez1, Kathrin Öhlenschläger1, Marc Freichel1 1Institute of Pharmacology, Ruprecht-Karls Heidelberg University JoVE 57222 면역학 및 감염병학 对离体乳头肌的心脏收缩力对小鼠的研究收缩尺寸 Sebastian Uhl1, Marc Freichel1, Ilka Mathar1 1Pharmakologisches Institut, Universität Heidelberg JoVE 53076 생물학
通过压力-体积环路分析确定心脏对β肾上腺素能刺激的反应 Rebekka Medert*1,2, Lucas Bacmeister*1,2,3, Sebastian Segin*1,2,4, Marc Freichel1,2, Juan E. Camacho Londoño1,2 1Pharmakologisches Institut, Ruprecht-Karls-Universität Heidelberg, 2DZHK (German Centre for Cardiovascular Research), partner site Heidelberg/Mannheim, 3Department of Internal Medicine III, University of Heidelberg, 4Department of Anesthesiology, University Hospital RWTH Aachen JoVE 62057 의학
腹膜源性肥大细胞的分离及其功能特征的 Ca2 +成像和脱粒测定 Volodymyr Tsvilovskyy1, Alejandra Solis-Lopez1, Kathrin Öhlenschläger1, Marc Freichel1 1Institute of Pharmacology, Ruprecht-Karls Heidelberg University JoVE 57222 면역학 및 감염병학
对离体乳头肌的心脏收缩力对小鼠的研究收缩尺寸 Sebastian Uhl1, Marc Freichel1, Ilka Mathar1 1Pharmakologisches Institut, Universität Heidelberg JoVE 53076 생물학